2.2.5 – Viruses are not living organisms and DO NOT multiply once inside a Host Cell

It is acknowledged by medical science that a virus is a nonliving particle, that has no eyes, no mouth, no nose, no orifices at all, no arms, no legs, no digestive system, and no reproductive system.

Viruses, like bacteria, are microscopic and cause human diseases. But unlike bacteria, viruses are acellular particles (meaning they aren’t made up of living cells like plants and animals are), consisting instead of a central core of either DNA or RNA surrounded by a coating of protein.

Viruses also lack the properties of living things: They have no energy metabolism, they do not grow, they produce no waste products, and they do not respond to stimuli. They also don’t reproduce independently but must replicate by invading living cells.

It is claimed, but never scientifically proven that it can multiply once inside a host cell.

There is not a single microscope video nor progressive slides of the virus moving or entering the cells of the human body or of it using the genetic material of a cell (once in the cell) to multiply itself and then cause cell damage / death and how it moves to other cells to do the same.

Further reading…

  1. https://www.news-medical.net/life-sciences/Are-Viruses-a-Lifeform.aspx

Thomas Cowan on “Virus”

“Most important, we will show that the minute particles called viruses are actually exosomes—not invaders but toxin-gobbling messengers that our cells produce to help us adjust to environmental assaults, including electro-smog. After all, most people have adjusted to worldwide radio waves, electricity in their homes, and ubiquitous Wi-Fi (and the sparrow population rebounded after the flu of 1738); exosomes are what allow this to happen. These tiny messengers provide real-time and rapid genetic adaptation to environmental changes. Whether these exosomes can help us adapt to the extreme disruption of 5G is the question of the day.”

Cowan, Thomas S.; Fallon Morell, Sally. The Truth About Contagion: Exploring Theories of How Disease Spreads (pp. 41-42). Skyhorse. Kindle Edition.

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